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Format: 147 S , 20 cm

Edition: 1. Aufl

Uniform Title: The castle of Otranto 〈dt.〉

Language: German

Subjects: English Studies

Keywords: Abenteuerroman ; Belletristische Darstellung ; Gesellschaftsroman

Author information: Walpole, Horace 1717-1797

Author information: Findeisen, Helmut 1926-2006

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Format: 224 S. , Ill., graph. Darst.

Edition: 2. Aufl.

ISBN: 3593349493

Uniform Title: Time management

Language: German

Subjects: Economics

Keywords: Zeiteinteilung ; Selbstmanagement ; Zeiteinteilung ; Führungskraft ; Ratgeber ; Ratgeber. ; Selbstmanagement ; Zeiteinteilung

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Format: 1 online resource (141 pages)

Edition: 1st ed.

ISBN: 9780975747551

Additional Edition: Print version: Baum, Scott Fault Lines Exposed Melbourne : Monash University Publishing,c2005 ISBN 9780975747544

Language: English

Keywords: Electronic books. ; Electronic books.

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Format: 1 online resource (139 pages)

ISBN: 9781135467449

Series Statement: Studies in American Popular History and Culture Series

Additional Edition: Print version: Slawinski, Scott Validating Bachelorhood Milton : Taylor & Francis Group,c2005 ISBN 9780415654746

Language: English

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Format: 1 online resource (254 pages)

Edition: 1st ed.

ISBN: 9783031258206

Series Statement: Nečas Center Series

Additional Edition: Print version: Scott, Jennifer Algorithms for Sparse Linear Systems Cham : Springer International Publishing AG,c2023 ISBN 9783031258190

Language: English

Keywords: Electronic books.

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Format: 1 online resource (374 pages)

Edition: 1st ed.

ISBN: 9789811511851

Note: Intro -- Preface -- Contents -- Part I: Basic Science of Pulmonary Development and Pulmonary Arterial Disease -- 1: Perspective for Part I -- 2: The Alveolar Stem Cell Niche of the Mammalian Lung -- 2.1 Introduction: The Alveolar Type 2 Epithelial Stem Cell Niche -- 2.2 Evidence for Heterogeneity in the AT2 Population -- 2.3 Signaling Pathways in the Stem Cell Niche -- 2.4 The Role of Immune Cells and Stromal Cells in Alveolar Repair and Regeneration -- 2.5 Future Directions and Clinical Implications -- References -- 3: Lung Development and Notch Signaling -- 3.1 Introduction -- 3.2 Morphogenesis and Epithelial Progenitors -- 3.3 Notch Signaling Controls Both Epithelial Cell Fates and Distributions -- 3.4 Development of NE Cell Clusters on Bifurcating Area of Branching Airways -- 3.5 Notch-Hes1 Signaling Is Required for Restricted Differentiation of Solitary NE Cells -- 3.6 Directional Migration of NE Cells Toward Bifurcation Points Creates Nodal NEBs -- References -- 4: Specialized Smooth Muscle Cell Progenitors in Pulmonary Hypertension -- 4.1 Introduction -- 4.2 Hypoxia-Induced Distal Pulmonary Arteriole SMCs Derive from Specialized SMC Progenitors -- 4.3 Stereotyped Program of Distal Muscularization -- 4.4 Monoclonal Expansion of SMCs in PH -- 4.5 Signaling Pathways Regulating Primed Cells -- 4.6 Future Direction and Clinical Implications -- References -- 5: Diverse Pharmacology of Prostacyclin Mimetics: Implications for Pulmonary Hypertension -- 5.1 Introduction -- 5.2 Development of Prostacyclin Mimetics and Their Diverse Pharmacology -- 5.3 Prostanoid Synthesis and Receptor Expression -- 5.3.1 Bronchial Smooth Muscle -- 5.3.2 Pulmonary Blood Vessels -- 5.3.2.1 Endothelium -- 5.3.2.2 Pulmonary Artery -- 5.3.2.3 Differential Prostanoid Expression in Distal Pulmonary Artery and Veins -- 5.3.2.4 Distal Pulmonary Veins. , 5.3.3 Prostanoid Receptor Expression in PAH -- 5.3.3.1 Downregulation of IP Receptors in PAH -- 5.3.3.2 Robust Expression of EP2 and EP4 Receptors in PAH: Key Anti-Fibrotic Targets -- 5.3.3.3 EP3 Receptors May Contribute to Disease Pathology in PAH -- 5.3.3.4 Role of the Veins in PAH and Other Classified Groups of PH -- 5.4 BMPR2 and TGF-β Signalling in PAH and Impact of Prostacyclin Analogues -- 5.5 Regulation of TASK-1 By Prostacyclin Mimetics: Implications in PAH -- 5.6 Prostacyclin Effects on Vascular Remodelling In Vivo: Outstanding Issues -- 5.7 Future Work and Clinical Implications -- References -- 6: Endothelial-to-Mesenchymal Transition in Pulmonary Hypertension -- 6.1 Pulmonary Hypertension -- 6.2 Endothelial-to-Mesenchymal Transition -- 6.3 EndoMT in PAH Pathogenesis -- 6.3.1 EndoMT in PAH Vascular Remodeling -- 6.3.2 Molecular Pathways of EndoMT in PAH -- 6.4 Conclusion -- 6.5 Future Direction and Clinical Implications -- References -- 7: Extracellular Vesicles, MicroRNAs, and Pulmonary Hypertension -- 7.1 Extracellular Vesicles (EV) -- 7.2 EV in Pulmonary Hypertension (PH) -- 7.3 MicroRNA Transfer Through EV in PH -- 7.4 Future Direction and Clinical Implications -- References -- 8: Roles of Tbx4 in the Lung Mesenchyme for Airway and Vascular Development -- References -- 9: A lacZ Reporter Transgenic Mouse Line Revealing the Development of Pulmonary Artery -- References -- 10: Roles of Stem Cell Antigen-1 in the Pulmonary Endothelium -- References -- 11: Morphological Characterization of Pulmonary Microvascular Disease in Bronchopulmonary Dysplasia Caused by Hyperoxia in Newborn Mice -- References -- 12: Involvement of CXCR4 and Stem Cells in a Rat Model of Pulmonary Arterial Hypertension -- References. , 13: Ca2+ Signal Through Inositol Trisphosphate Receptors for Cardiovascular Development and Pathophysiology of Pulmonary Arterial Hypertension -- References -- Part II: Abnormal Pulmonary Circulation in the Developing Lung and Heart -- 14: Perspective for Part II -- 14.1 Idiopathic Pulmonary Arterial Hypertension (IPAH) -- 14.2 Pulmonary Hypertension with Congenital Heart Disease -- 14.3 Pulmonary Circulation in Patients with Congenital Heart Disease -- References -- 15: Pathophysiology of Pulmonary Circulation in Congenital Heart Disease -- 15.1 Introduction -- 15.2 Comprehensive Assessment of Integrated Pulmonary Circulation -- 15.2.1 Physiologic Components of Pulmonary Circulation -- 15.2.2 Impedance Analysis -- 15.3 Pathophysiological Characteristics of Pulmonary Circulation in Congenital Heart Disease -- 15.3.1 Abnormal Resistance Is the Main Pathophysiology -- 15.3.2 Right Ventricular Function and Coupling to PA Load -- 15.3.3 Abnormalities of Compliance Is the Main Pathophysiology -- 15.3.4 Non-pulsatile Pulmonary Flow Is the Main Pathophysiology -- References -- 16: Development of Novel Therapies for Pulmonary Hypertension by Clinical Application of Basic Research -- 16.1 Introduction -- 16.2 Endothelial Function in the Development of PAH -- 16.3 PASMCs in the Development of PAH -- 16.4 Selenoprotein P in the Development of PAH -- 16.5 Conclusion -- References -- 17: Using Patient-Specific Induced Pluripotent Stem Cells to Understand and Treat Pulmonary Arterial Hypertension -- 17.1 Introduction -- 17.2 Patient-Specific iPSC-Derived Endothelial Cells to Model PAH -- 17.2.1 iPSC-EC Recapitulates Native Pulmonary Arterial Endothelial Cell (PAEC) -- 17.2.2 Patient-Specific Drug Response in IPSC-EC and PAEC -- 17.3 Modeling Reduced Penetrance of BMPR2 Mutation in PAH. , 17.3.1 Preserved EC Function in Unaffected BMPR2 Mutation Carrier (UMC) -- 17.3.2 Preserved pP38 Signaling Pathway in Unaffected BMPR2 Mutation Carrier -- 17.4 Gene Editing in PAH IPSCs -- 17.4.1 Correction of the BMPR2 Mutation in PAH iPSCs -- 17.4.2 Generation of iPSC Line with BMPR2 Mutation -- 17.5 Future Directions and Clinical Implications -- References -- 18: Modeling Pulmonary Arterial Hypertension Using Induced Pluripotent Stem Cells -- 18.1 Heritable Pulmonary Arterial Hypertension -- 18.1.1 Insights into the Pathobiology of PAH -- 18.1.2 Reduced Penetrance of BMPR2 in PAH -- 18.2 Modeling Pulmonary Arterial Hypertension with Induced Pluripotent Stem Cells -- 18.2.1 Embryological Origins of the Pulmonary Vasculature -- 18.2.2 Current iPSC Models of PAH -- 18.3 Future Direction and Clinical Implications -- References -- 19: Dysfunction and Restoration of Endothelial Cell Communications in Pulmonary Arterial Hypertension: Therapeutic Implications -- 19.1 Introduction -- 19.2 Pulmonary Endothelial Dysfunction and the Pathobiology of PAH -- 19.3 Current Promising Strategies for Restoring Pulmonary Endothelial Dysfunction and Cell-Cell Communications -- 19.3.1 Restoring the Balance of Vasodilation and Vasoconstriction -- 19.3.2 Restitution of the Defective BMPR-2 Signaling System -- 19.3.3 Targeting Cell Proliferation and Cell accumulation -- 19.3.4 Restitution of an Adapted Extracellular Matrix (ECM) Remodeling -- 19.3.5 Targeting Metabolic Changes -- 19.3.6 Targeting the Vicious Cycle Between Endothelial Dysfunction and Immune Dysregulation -- 19.4 Future Directions and Clinical Implications -- References -- 20: Inflammatory Cytokines in the Pathogenesis of Pulmonary Arterial Hypertension -- 20.1 Background -- 20.2 IL-6 in the Pathogenesis of HPH -- 20.3 IL-21 in the Pathogenesis of HPH. , 20.4 Increased Expression of IL-21 and M2 Macrophage Markers in the Lungs of IPAH Patients -- References -- 21: Genotypes and Phenotypes of Chinese Pediatric Patients with Idiopathic and Heritable Pulmonary Arterial Hypertension: Experiences from a Single Center -- 21.1 Introduction -- 21.2 Methods -- 21.3 Selection of Patients -- 21.4 Genetic Studies -- 21.5 Statistical Analysis -- 21.6 Results -- 21.6.1 Clinical Characteristics -- 21.6.2 Targeted Drug Therapy -- 21.6.3 Outcome of Patients -- 21.7 Discussion -- References -- 22: Fundamental Insight into Pulmonary Vascular Disease: Perspectives from Pediatric PAH in Japan -- 22.1 Early Detection and Early Treatment of PAH: Mechanistic Insights -- 22.2 Pathological Basis of Atypical CHD-PAH: Clinical and Mechanistic Implications -- 23: Risk Stratification in Paediatric Pulmonary Arterial Hypertension -- 23.1 Why Risk Stratify? -- 23.2 Multidimensional Risk Stratification -- 23.3 Factors to Consider in Multidimensional Risk Stratification of children with Pulmonary Arterial Hypertension -- 23.4 Cause of Pulmonary Hypertension -- 23.5 Vascular Burden -- 23.6 Ventricular Function -- 23.7 Impact on the Patient -- 23.8 Summary -- References -- 24: The Adaptive Right Ventricle in Eisenmenger Syndrome: Potential Therapeutic Targets for Pulmonary Hypertension? -- 24.1 Introduction -- 24.2 Improved Survival in Eisenmenger Syndrome -- 24.3 Preserved Fetal Morphology in Eisenmenger Syndrome -- 24.4 Fetal Phenotype in Ovine CHD Model -- 24.5 The Adaptive RV Response to Acute Afterload-RV Anrep Effect -- 24.6 Potential Mechanisms of RV Anrep Effect -- 24.7 Future Directions and Clinical Implications -- References -- 25: Impaired Right Coronary Vasodilator Function in Pulmonary Hypertensive Rats Assessed by In Vivo Synchrotron Microangiography -- References. , 26: Relationship Between Mutations in ENG and ALK1 Genes and the Affected Organs in Hereditary Hemorrhagic Telangiectasia.

Additional Edition: Print version: Nakanishi, Toshio Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension Singapore : Springer Singapore Pte. Limited,c2020 ISBN 9789811511844

Language: English

Keywords: Electronic books. ; Congress ; Conference papers and proceedings. ; Conference papers and proceedings. ; Actes de congrès.

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Format: 1 online resource (220 pages)

Edition: 1st ed.

ISBN: 9783031348044

Series Statement: The International Library of Ethics, Law and Technology Series ; v.40

Note: Intro -- Contents -- Chapter 1: Introduction: Understanding and Regulating AI-Powered Recommender Systems -- References -- Part I: Fairness and Transparency -- Chapter 2: Recommender Systems and Discrimination -- 2.1 Introduction -- 2.2 Reasons for Discriminating Recommendations -- 2.2.1 Lack of Diversity in Training Data -- 2.2.2 (Unconscious) Bias in Training Data -- 2.2.3 Modelling Algorithm -- 2.2.4 Interim Conclusion and Thoughts -- 2.3 Legal Frame -- 2.3.1 Agreement - Data Protection Law -- 2.3.2 Information - Unfair Competition Law -- 2.3.3 General Anti-discrimination Law -- 2.3.4 Interim Conclusion -- 2.4 Outlook -- 2.4.1 Extreme Solutions -- 2.4.2 Further Development of the Information Approach -- 2.4.3 Monitoring and Audit Obligations -- 2.4.4 Interim Conclusion and Thoughts -- 2.5 Conclusions -- References -- Chapter 3: From Algorithmic Transparency to Algorithmic Choice: European Perspectives on Recommender Systems and Platform Regulation -- 3.1 Introduction -- 3.2 Recommender Governance in the EU Platform Economy -- 3.2.1 Mapping the Regulatory Landscape -- 3.2.2 Layers of Terminology in EU Law: "Rankings" and "Recommender Systems" -- 3.3 Five Axes of Algorithmic Transparency: A Comparative Analysis -- 3.3.1 Purpose of Transparency -- 3.3.2 Audiences of Disclosure -- 3.3.3 Addressees of the Duty to Disclose -- 3.3.4 Content of the Disclosure -- 3.3.5 Modalities of Disclosure -- 3.4 The Digital Services Act: From Algorithmic Transparency to Algorithmic Choice? -- 3.4.1 Extension of Transparency Rules -- 3.4.2 User Control Over Ranking Criteria -- 3.5 Third Party Recommender Systems: Towards a Market for "RecommenderTech" -- 3.6 Conclusion -- References -- Chapter 4: Black Hole Instead of Black Box?: The Double Opaqueness of Recommender Systems on Gaming Platforms and Its Legal Implications -- 4.1 Introduction. , 4.2 The Black Box-Problem of AI Applications -- 4.2.1 Transparency and Explainability: An Introduction -- 4.2.2 Efficiency vs. Explainability of Machine Learning -- 4.2.3 Background of the Transparency Requirement -- 4.2.4 Criticism -- 4.2.5 In Terms of Recommender Systems -- 4.3 The Black Hole-Problem of Gaming Platforms -- 4.3.1 Types of Recommender Systems -- 4.3.1.1 Content-Based Filtering Methods -- 4.3.1.2 Collaborative Filtering Methods -- 4.3.1.3 Hybrid Filtering Methods -- 4.3.2 Black Hole Phenomenon -- 4.4 Legal Bases and Consequences -- 4.4.1 Legal Acts -- 4.4.2 Digital Services Act -- 4.4.2.1 Problem Description -- 4.4.2.2 Regulatory Content Related to Recommender Systems -- 4.4.3 Artificial Intelligence Act -- 4.4.3.1 Purpose of the Draft Act -- 4.4.3.2 Regulatory Content Related to Recommender Systems -- 4.4.4 Dealing with Legal Requirements -- 4.4.4.1 User-Oriented Transparency -- 4.4.4.2 Government Oversight -- 4.4.4.3 Combination of the Two Approaches with Additional Experts -- 4.5 Implementation of the Proposed Solutions -- 4.5.1 Standardization -- 4.5.2 Control Mechanisms -- 4.6 Conclusion -- References -- Chapter 5: Digital Labor as a Structural Fairness Issue in Recommender Systems -- 5.1 Introduction: Multisided (Un)Fairness in Recommender Systems -- 5.2 Digital Labor as a Structural Issue in Recommender Systems -- 5.3 Fairness Issues from Value Distribution to Work Conditions and Laborers' Awareness -- 5.4 Addressing the Problem -- 5.5 Conclusion -- References -- Part II: Manipulation and Personal Autonomy -- Chapter 6: Recommender Systems, Manipulation and Private Autonomy: How European Civil Law Regulates and Should Regulate Recommender Systems for the Benefit of Private Autonomy -- 6.1 Introduction -- 6.2 Autonomy and Influence in Private Law -- 6.3 Recommender Systems and Their Influence -- 6.4 Manipulation. , 6.5 Recommender Systems and Manipulation -- 6.5.1 Recommendations in General -- 6.5.2 Labelled Recommendations -- 6.5.3 Unrelated Recommendations -- 6.5.3.1 In General -- 6.5.3.2 Targeted Recommendations -- 6.5.3.2.1 In General -- 6.5.3.2.2 Exploiting Emotions -- 6.5.3.2.3 Addressing Fears Through (Allegedly) Harm-Alleviating Offers -- 6.5.4 Interim Conclusion: Recommender Systems, Manipulation and Private Autonomy -- 6.6 Regulation Regarding Recommender Systems -- 6.6.1 Unexpected Recommendation Criteria -- 6.6.2 Targeted Recommendations Exploiting Emotions or Addressing Fears -- 6.6.3 Regulative Measures to Take Regarding Recommender Systems -- 6.7 Conclusion -- References -- Chapter 7: Reasoning with Recommender Systems? Practical Reasoning, Digital Nudging, and Autonomy -- 7.1 Introduction -- 7.2 Practical Reasoning, Choices, and Recommendations -- 7.3 Recommender Systems and Digital Nudging -- 7.4 Autonomy in Practical Reasoning with Recommender Systems -- 7.5 Conclusion -- References -- Chapter 8: Recommending Ourselves to Death: Values in the Age of Algorithms -- 8.1 Introduction -- 8.2 Distorting Forces -- 8.2.1 Past Evaluative Standards -- 8.2.2 Reducing to Computable Information -- 8.2.3 Proxies for 'Good' -- 8.2.4 Black Boxed -- 8.3 Changing Human Values -- 8.4 Same Problem with Humans? -- 8.5 Conclusion -- References -- Part III: Designing and Evaluating Recommender Systems -- Chapter 9: Ethical and Legal Analysis of Machine Learning Based Systems: A Scenario Analysis of a Food Recommender System -- 9.1 Introduction -- 9.2 An Example Application: FoodApp- the Application for Meal Delivery -- 9.3 Current Approaches to Ethical Analysis of Recommender Systems -- 9.4 Ethical Analysis -- 9.5 Legal Considerations -- 9.5.1 Data Protection Law -- 9.5.2 General Principles and Lawfulness of Processing Personal Data -- 9.5.3 Lawfulness. , 9.5.4 Purpose Limitation and Access to Data -- 9.5.5 Data Minimization and Storage Limitation -- 9.5.6 Accuracy, Security and Impact Assessment -- 9.6 Results of the Combined Ethical and Legal Analysis Approach -- 9.7 Conclusion and Outlook -- References -- Chapter 10: Factors Influencing Trust and Use of Recommendation AI: A Case Study of Diet Improvement AI in Japan -- 10.1 Society 5.0 and Recommendation AI in Japan -- 10.2 Model for Ensuring Trustworthiness of AI Services -- 10.3 Components of a Trustworthy AI Model -- 10.3.1 AI Intervention -- 10.3.2 Data Management -- 10.3.3 Purpose of Use -- 10.4 Verification of Trustworthy AI Model: A Case Study of AI for Dietary Habit Improvement Recommendations -- 10.4.1 Subjects -- 10.4.2 Verification 1: AI Intervention -- 10.4.3 Verification 2: Data Management -- 10.4.4 Verification 3: Purpose of Use -- 10.4.5 Method -- 10.4.6 Results -- 10.4.6.1 AI Intervention -- 10.4.6.2 Data Management -- 10.4.6.3 Purpose of Use in Terms of Service Agreements -- 10.5 Necessary Elements for Trusted AI -- References -- Chapter 11: Ethics of E-Learning Recommender Systems: Epistemic Positioning and Ideological Orientation -- 11.1 Introduction -- 11.2 Methods of Recommender Systems -- 11.3 Recommender Systems in e-Learning -- 11.3.1 Filtering Techniques: What Implications on Social and Epistemic Open-Mindedness? -- 11.3.2 Model Selection: A Risk of Thinking Homogenization? -- 11.3.3 Assessment Methods: What Do They Value? -- 11.4 Problem Statement -- 11.5 Some Proposals -- 11.5.1 Knowledge-Based Recommendations -- 11.5.2 A Learner Model Coming from Cognitive and Educational Sciences -- 11.5.3 A Teaching Model Based on Empiric Analyses -- 11.5.4 Explainable Recommendations -- 11.6 Discussion and Conclusion -- References.

Additional Edition: Print version: Genovesi, Sergio Recommender Systems: Legal and Ethical Issues Cham : Springer International Publishing AG,c2023 ISBN 9783031348037

Language: English

Keywords: Electronic books. ; Electronic books.

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Format: 1 online resource (xiii, 405 pages)

ISBN: 9780203995549 , 9781134882694 , 9781134882731 , 9781134882748

Content: BOOK COVER; HALF-TITLE; TITLE; COPYRIGHT; CONTENTS; FIGURES; TABLES; CONTRIBUTORS; PREFACE; ACKNOWLEDGEMENTS; 1 INDUSTRIALIZATION AND REGIONAL DEVELOPMENT; 2 FORDIST AND POST-FORDIST INTERNATIONAL DIVISION OF LABOR AND MONETARY REGIMES; 3 FORDISM AND POST-FORDISM: A CRITICAL REFORMULATION; 4 FLEXIBLE SPECIALIZATION VERSUS POST-FORDISM: THEORY, EVIDENCE, AND POLICY IMPLICATIONS; 5 THE JAPANESE MODEL OF POST-FORDISM; 6 THE REVITALIZATION OF MASS PRODUCTION IN THE COMPUTER AGE; 7 TECHNOLOGICAL TRAJECTORIES AND THE CLASSICAL REVIVAL IN ECONOMICS.

Note: Includes bibliographical references (pages 349-401) and index

Additional Edition: ISBN 9780415087520

Additional Edition: Erscheint auch als Druck-Ausgabe ISBN 9780415087520

Language: English

Keywords: Electronic books ; Conference papers and proceedings.

DOI: 10.4324/9780203995549

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URL: Full text available: 1992.

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Format: 128 S , Ill., graph. Darst , 24 cm

ISBN: 3706401738

Series Statement: New business line 49

Uniform Title: Empowerment 〈dt.〉

Language: German

Keywords: Management by Participation ; Ratgeber - Formschlagwort. ; Empowerment ; Führungstechnik ; Kooperativer Führungsstil ; Management by participation

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UID:

Format: 178 S.

Edition: 1. Aufl.

Series Statement: bb 452

Uniform Title: Payment deferred 〈dt.〉

Language: German

Subjects: English Studies

Keywords: Belletristische Darstellung

Author information: Forester, Cecil S. 1899-1966