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Expert System for Drying Schemes of Agricultural Products Based on Fuzzy Control and BP Neural Network

Jing Huang, Jing Huang ; Jing Huang, Wei Xiong ; Wei Xiong, Honglei Xu ; [et al.]

Angle Publishing Co., Ltd. ; 2021

In: 網際網路技術學刊 Vol. 22, No. 6 ( 2021-11), p. 1253-1263

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In: 網際網路技術學刊, Angle Publishing Co., Ltd., Vol. 22, No. 6 ( 2021-11), p. 1253-1263

Type of Medium: Online Resource

ISSN: 1607-9264 , 1607-9264

Uniform Title: Expert System for Drying Schemes of Agricultural Products Based on Fuzzy Control and BP Neural Network

URL: Article

DOI: 10.53106/160792642021112206005

Language: Unknown

Publisher: Angle Publishing Co., Ltd.

Publication Date: 2021

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Lowering systolic blood pressure to less than 120 mm Hg versus less than 140 mm Hg in patients with high cardiovascular risk with and without diabetes or previous stroke: an open-label, blinded-outcome, randomised trial

Liu, Jiamin ; Li, Yan ; Ge, Jinzhuo ; [et al.]

Elsevier BV ; 2024

In: The Lancet Vol. 404, No. 10449 ( 2024-07), p. 245-255

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In: The Lancet, Elsevier BV, Vol. 404, No. 10449 ( 2024-07), p. 245-255

Type of Medium: Online Resource

ISSN: 0140-6736

URL: Article

DOI: 10.1016/S0140-6736(24)01028-6

RVK:

XA 10000

Language: English

Publisher: Elsevier BV

Publication Date: 2024

detail.hit.zdb_id: 2067452-1

detail.hit.zdb_id: 3306-6

detail.hit.zdb_id: 1476593-7

SSG: 5,21

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Nonstructural protein 2A2 from Duck hepatitis A virus type 1 inhibits interferon beta production by interaction with mitochondrial antiviral signaling protein and TANK-binding kinase 1

Sui, Nana ; Zhang, Ruihua ; Jiang, Yue ; [et al.]

Elsevier BV ; 2023

In: Veterinary Microbiology Vol. 280 ( 2023-05), p. 109679-

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In: Veterinary Microbiology, Elsevier BV, Vol. 280 ( 2023-05), p. 109679-

Type of Medium: Online Resource

ISSN: 0378-1135

URL: Article

DOI: 10.1016/j.vetmic.2023.109679

Language: English

Publisher: Elsevier BV

Publication Date: 2023

detail.hit.zdb_id: 1498996-7

SSG: 22

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Table_9.xls

Sui, Nana ; Zhang, Ruihua ; Jiang, Yue ; [et al.]

Frontiers Media SA ; 2022

In: Frontiers in Cellular and Infection Microbiology Vol. 12 ( 2022-4-22)

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In: Frontiers in Cellular and Infection Microbiology, Frontiers Media SA, Vol. 12 ( 2022-4-22)

Abstract: Duck hepatitis A virus type 1 (DHAV-1) is a highly lethal virus that severely affects the duck industry worldwide. Long noncoding RNAs (lncRNAs) exert crucial roles in pathogen attacks. Here, we conducted deep transcriptome analysis to investigate the dynamic changes of host lncRNAs profiles in DHAV-1-infected duck embryo fibroblasts. We identified 16,589 lncRNAs in total and characterized their genomic features. Moreover, 772 and 616 differentially expressed lncRNAs (DELs) were screened at 12 and 24 h post-infection. Additionally, we predicted the DELs’ cis- and trans-target genes and constructed lncRNA-target genes regulatory networks. Functional annotation analyses indicated that the putative target genes of DELs participated in diverse vital biological processed, including immune responses, cellular metabolism, and autophagy. For example, we confirmed the dysregulation of pattern recognition receptors (TLR3, RIG-I, MDA5, LGP2, cGAS), signal transducers (STAT1), transcription factors (IRF7), immune response mediators (IL6, IL10, TRIM25, TRIM35, TRIM60, IFITM1, IFITM3, IFITM5), and autophagy-related genes (ULK1, ULK2, EIF4EBP2) using RT-qPCR. Finally, we confirmed that one DHAV-1 induced lncRNA-XR_003496198 is likely to inhibit DHAV-1 replication in DEFs. Our study comprehensively analyzed the lncRNA profiles upon DHAV-1 infection and screened the target genes involved in the innate immune response and autophagy signaling pathway, thereby revealing the essential roles of duck lncRNAs and broadening our understanding of host-virus interactions.

Type of Medium: Online Resource

ISSN: 2235-2988

URL: Article

DOI: 10.3389/fcimb.2022.858537

DOI: 10.3389/fcimb.2022.858537.s001

DOI: 10.3389/fcimb.2022.858537.s002

DOI: 10.3389/fcimb.2022.858537.s003

DOI: 10.3389/fcimb.2022.858537.s004

DOI: 10.3389/fcimb.2022.858537.s005

DOI: 10.3389/fcimb.2022.858537.s006

DOI: 10.3389/fcimb.2022.858537.s007

DOI: 10.3389/fcimb.2022.858537.s008

DOI: 10.3389/fcimb.2022.858537.s009

DOI: 10.3389/fcimb.2022.858537.s010

Language: Unknown

Publisher: Frontiers Media SA

Publication Date: 2022

detail.hit.zdb_id: 2619676-1

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Table_7.xlsx

Sui, Nana ; Zhang, Ruihua ; Jiang, Yue ; [et al.]

Frontiers Media SA ; 2022

In: Frontiers in Cellular and Infection Microbiology Vol. 11 ( 2022-1-3)

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In: Frontiers in Cellular and Infection Microbiology, Frontiers Media SA, Vol. 11 ( 2022-1-3)

Abstract: Duck hepatitis A virus 1 (DHAV-1) is a highly contagious etiological agent that causes acute hepatitis in young ducklings. MicroRNAs (miRNAs) play important regulatory roles in response to pathogens. However, the interplay between DHAV-1 infection and miRNAs remains ambiguous. We characterized and compared miRNA and mRNA expression profiles in duck embryo fibroblasts cells (DEFs) infected with DHAV-1. In total, 36 and 96 differentially expressed (DE) miRNAs, and 4110 and 2595 DE mRNAs, were identified at 12 and 24 h after infection. In particular, 126 and 275 miRNA–mRNA pairs with a negative correlation were chosen to construct an interaction network. Subsequently, we identified the functional annotation of DE mRNAs and target genes of DE miRNAs enriched in diverse Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathways, which may be important for virus resistance, cell proliferation, and metabolism. Moreover, upregulated miR-222a could negatively regulate DHAV-1 replication in DEFs and downregulate integrin subunit beta 3 (ITGB3) expression by targeting the 3′ untranslated region (3′UTR), indicating that miR-222a may modulate DHAV-1 replication via interaction with ITGB3. In conclusion, the results reveal changes of mRNAs and miRNAs during DHAV-1 infection and suggest miR-222a as an antiviral factor against DHAV-1.

Type of Medium: Online Resource

ISSN: 2235-2988

URL: Article

DOI: 10.3389/fcimb.2021.811556

DOI: 10.3389/fcimb.2021.811556.s001

DOI: 10.3389/fcimb.2021.811556.s002

DOI: 10.3389/fcimb.2021.811556.s003

DOI: 10.3389/fcimb.2021.811556.s004

DOI: 10.3389/fcimb.2021.811556.s005

DOI: 10.3389/fcimb.2021.811556.s006

DOI: 10.3389/fcimb.2021.811556.s007

DOI: 10.3389/fcimb.2021.811556.s008

DOI: 10.3389/fcimb.2021.811556.s009

DOI: 10.3389/fcimb.2021.811556.s010

DOI: 10.3389/fcimb.2021.811556.s011

DOI: 10.3389/fcimb.2021.811556.s012

DOI: 10.3389/fcimb.2021.811556.s013

DOI: 10.3389/fcimb.2021.811556.s014

DOI: 10.3389/fcimb.2021.811556.s015

DOI: 10.3389/fcimb.2021.811556.s016

DOI: 10.3389/fcimb.2021.811556.s017

DOI: 10.3389/fcimb.2021.811556.s018

DOI: 10.3389/fcimb.2021.811556.s019

DOI: 10.3389/fcimb.2021.811556.s020

DOI: 10.3389/fcimb.2021.811556.s021

Language: Unknown

Publisher: Frontiers Media SA

Publication Date: 2022

detail.hit.zdb_id: 2619676-1

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