UID:
almahu_9948025474802882
Format:
1 online resource (443 p.)
Edition:
3rd ed.
ISBN:
1-282-12086-7
,
9786612120862
,
0-08-092118-3
Content:
Hypertensive disorders remain one the major causes of maternal and fetal morbidity and death. It is also a leading cause of preterm birth now known to be a risk factor in remote cardiovascular disease. Despite this, the hypertensive disorders remain marginally studied, and their management is commonly controversial. Chesley's Hypertensive Disorders in Pregnancy remains one of the beacons to guide this field, recognized for its uniqueness and utility.
Note:
Description based upon print version of record.
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Front Cover; CHESLEY'S HYPERTENSIVE DISORDERS IN PREGNANCY; COPYRIGHT; Contents; List of Contributors; Preface; CHAPTER 1 Introduction, History, Controversies, and Definitions; HISTORY; SIGNS; HYPOTHESES AND RATIONAL MANAGEMENT; PROPHYLAXIS; CLASSIFICATION OF THE HYPERTENSIVE DISORDERS IN PREGNANCY; EDITORS' UPDATE; DENOUEMENT; References; CHAPTER 2 The Clinical Spectrum of Preeclampsia; INTRODUCTION; CLINICAL MANIFESTATIONS OF PREECLAMPSIA SYNDROME; DIFFERENTIAL DIAGNOSIS; References; CHAPTER 3 Epidemiology of Pregnancy-Related Hypertension; FREQUENCY OF OCCURRENCE
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DISCUSSION OF DIFFERENTIAL FREQUENCY ESTIMATESRISK FACTORS; CLINICAL PREDICTORS; FIRST BIRTH AND OTHER PLACENTAL FACTORS; NATURAL HISTORY; CRITIQUE OF STUDIES; CONCLUSION; References; CHAPTER 4 Genetic Factors in the Etiology of Preeclampsia/Eclampsia; DEDICATION; INTRODUCTION; BIOLOGICAL PATHWAYSOF PREECLAMPSIA; TYPES OF GENETIC STUDIES CONDUCTED; PROBLEMS WITH STUDYING THE GENETICS OF PREECLAMPSIA; A GENOMICS APPROACH TO PREECLAMPSIA; ESSENTIAL VARIABLES TO CONSIDER; THE FUTURE OF PREECLAMPSIA GENETIC RESEARCH; CONCLUSIONS; References
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CHAPTER 5 The Placenta in Normal Pregnancy and PreeclampsiaINTRODUCTION; THE MICROANATOMY OF NORMAL HUMAN PLACENTATION; THE MICROANATOMY OF ABNORMAL HUMAN PLACENTATION IN PREECLAMPSIA; THE ROAD TO PREECLAMPSIA; OXYGEN TENSION REGULATES HUMAN CYTOTROPHOBLAST PROLIFERATION AND DIFFERENTIATION IN VITRO; DURING NORMAL PREGNANCY, INVASIVE CYTOTROPHOBLASTS MODULATE THEIR ADHESION MOLECULE REPERTOIRE TO MIMIC THAT OF VASCULAR CELLS; IN PREECLAMPSIA, INVASIVE CYTOTROPHOBLASTS FAIL TO SWITCH THEIR ADHESION MOLECULE REPERTOIRE TO MIMIC THAT OF VASCULAR CELLS
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NOVEL UNBIASED APPROACHES FOR ADDRESSING THE COMPLEXITIES OF THE PREECLAMPSIA SYNDROMESUMMARY AND FUTURE DIRECTIONS; References; CHAPTER 6 Angiogenesis and Preeclampsia; INTRODUCTION; PLACENTALVASCULAR DEVELOPMENT IN HEALTH; ANGIOGENIC IMBALANCE IN PREECLAMPSIA; PERSPECTIVES; References; CHAPTER 7 Metabolic Syndrome and Preeclampsia; INTRODUCTION; METABOLIC SYNDROME; METABOLIC SYNDROME AND CARDIOVASCULAR DISEASE; PREGNANCY-INDUCED CHANGES IN INSULIN AND LIPID METABOLISM; PREECLAMPSIA AND METABOLIC SYNDROME; METABOLIC SYNDROME: A CAUSE OF PLACENTAL DYSFUNCTION?; SUMMARYAND PERSPECTIVES
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ReferencesCHAPTER 8 Immunology of Normal Pregnancy and Preeclampsia; INTRODUCTION; MATERNAL IMMUNE PRIMING FOR PREGNANCY- ADAPTING TO A FOREIGN FETUS; INNATE AND ADAPTIVE IMMUNITY; THE SYSTEMIC INFLAMMATORY NETWORK AND RESPONSE; ENDOTHELIAL CELLS ARE INFLAMMATORY CELLS; CYTOKINES, CHEMOKINES, GROWTH FACTORS, ADIPOKINES, AND ANGIOGENIC FACTORS; ACUTE PHASE RESPONSE; METABOLISM AND THE SYSTEMIC INFLAMMATORY RESPONSE; PREECLAMPSIA IS ATWO-STAGE DISEASE; MATERNAL-FETAL IMMUNE INTERFACES AND MATERNAL RECOGNITION OF FETAL (PATERNAL) ANTIGENS
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STAGE 1 PREECLAMPSIA, INTERFACE 1, AND MATERNAL IMMUNE RESPONSES TO TROPHOBLAST
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English
Additional Edition:
ISBN 0-12-374213-7
Language:
English
Keywords:
Aufsatzsammlung
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