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  • 1
    Language: English
    In: Science (New York, N.Y.), 09 March 2018, Vol.359(6380), pp.1100-1101
    Description: Communication in networks is the basis of many social and biological functions. Recent findings have added an uncomfortable twist to this view: Tumors can function as communicating networks, too. In several malignancies such as incurable brain tumors, long protrusions extend from cancer cells, connecting...
    Keywords: Cell Communication ; Brain Neoplasms -- Pathology ; Cell Surface Extensions -- Pathology ; Glioblastoma -- Pathology
    ISSN: 00368075
    E-ISSN: 1095-9203
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  • 2
    Language: English
    In: European Journal of Cancer, Sept, 2011, Vol.47, p.S357-S358
    Description: To link to full-text access for this article, visit this link: http://dx.doi.org/10.1016/S0959-8049(11)70202-7 Byline: Wolfgang Wick Author Affiliation: Department of Neurooncology, University of Heidelberg, Heidelberg, Germany
    Keywords: Gliomas
    ISSN: 0959-8049
    Source: Cengage Learning, Inc.
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  • 3
    Language: English
    In: European Journal of Cancer, Sept, 2011, Vol.47, p.S357-S358
    Description: To link to full-text access for this article, visit this link: http://dx.doi.org/10.1016/S0959-8049(11)70202-7 Byline: Wolfgang Wick Author Affiliation: Department of Neurooncology, University of Heidelberg, Heidelberg, Germany
    Keywords: Gliomas
    ISSN: 0959-8049
    Source: Cengage Learning, Inc.
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  • 4
    In: International Journal of Cancer, 15 June 2014, Vol.134(12), pp.2991-2992
    Description: To purchase or authenticate to the full-text of this article, please visit this link: http://onlinelibrary.wiley.com/doi/10.1002/ijc.28614/abstract Byline: Wolfgang Wick, Antje Wick, Michael Platten ***** No abstract is available for this article. ***** Article Note: Conflict of interest: W.W. reports on having received consulting and lecture fees from MSD, Roche and Magforce. W.W. has received research support from Apogenix, Boehringer Ingelheim, Eli Lilly, MSD, and Roche. He serves on the Steering Committee of the AVAglio trial involving bevacizumab in glioblastoma. A.W. and M.P. have no conflicts to report.
    Keywords: Glioblastomas;
    ISSN: 0020-7136
    E-ISSN: 1097-0215
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  • 5
    Language: English
    In: European Journal of Cancer, 2011, Vol.47, pp.S357-S358
    Keywords: Medicine
    ISSN: 0959-8049
    E-ISSN: 1879-0852
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  • 6
    Language: English
    In: Clinical cancer research : an official journal of the American Association for Cancer Research, 15 January 2018, Vol.24(2), pp.256-258
    Description: Glioblastoma has a gigantic unmet medical need. Molecular knowledge has evolved substantially, including data on clonal selection with progression. Past trials for all-comers may have produced false negative results. Molecular precision at progression needs workup of new tissue, and revisiting drugs with a focus on brain tumor penetration may yield surprises. .
    Keywords: Brain Neoplasms ; Glioblastoma
    ISSN: 1078-0432
    E-ISSN: 15573265
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  • 7
    Language: English
    In: best practice onkologie, May, 2018, Vol.13(3), p.152(4)
    ISSN: 0946-4565
    E-ISSN: 18628559
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  • 8
    Language: English
    In: Lancet Oncology, 2012, Vol.13(8), p.e329
    Description: To link to full-text access for this article, visit this link: http://dx.doi.org/10.1016/S1470-2045(12)70333-9 Byline: Wolfgang Wick (a), Michael Weller (b) Author Affiliation: (a) Department of Neurooncology, Neurology Clinic and National Centre for Tumour Disease, University of Heidelberg, D-69120 Heidelberg, Germany (b) Department of Neurology, University Hospital Zurich, Zurich, Switzerland
    Keywords: Elderly
    ISSN: 1470-2045
    Source: Cengage Learning, Inc.
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  • 9
    In: Nature, 2011, Vol.478(7368), p.197
    Description: Activation of the aryl hydrocarbon receptor (AHR) by environmental xenobiotic toxic chemicals, for instance 2,3,7,8-tetrachlorodibenzo-p-dioxin (dioxin), has been implicated in a variety of cellular processes such as embryogenesis, transformation, tumorigenesis and inflammation. But the identity of an endogenous ligand activating the AHR under physiological conditions in the absence of environmental toxic chemicals is still unknown. Here we identify the tryptophan (Trp) catabolite kynurenine (Kyn) as an endogenous ligand of the human AHR that is constitutively generated by human tumour cells via tryptophan-2,3-dioxygenase (TDO), a liver- and neuron-derived Trp-degrading enzyme not yet implicated in cancer biology. TDO-derived Kyn suppresses antitumour immune responses and promotes tumour-cell survival and motility through the AHR in an autocrine/paracrine fashion. The TDO-AHR pathway is active in human brain tumours and is associated with malignant progression and poor survival. Because Kyn is produced during cancer progression and inflammation in the local microenvironment in amounts sufficient for activating the human AHR, these results provide evidence for a previously unidentified pathophysiological function of the AHR with profound implications for cancer and immune biology.
    Keywords: Sciences (General) ; Physics;
    ISSN: 0028-0836
    E-ISSN: 14764687
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  • 10
    Language: English
    In: CNS oncology, November 2013, Vol.2(6), pp.491-3
    Description: Professor Wolfgang Wick speaks to Tess O'Neill, Head of Commissioning: Wolfgang Wick is the Chairman of the Department of Neuro-Oncology, Hertie Professor of Neuro-Oncology and Director at the National Tumor Center at the University of Heidelberg, Germany. He is conducting multicenter Phase III randomized trials for the Neuro-Oncology Working Group of the German Cancer Society, the European Organisation for Research and Treatment of Cancer as well as a number of multicenter trials with the pharmaceutical industry. He is a steering committee member of the Neuro-Oncology Working Group and the European Association for Neuro-oncology as well as chairman of the European Organisation for Research and Treatment of Cancer Brain Tumor Group. His main scientific interests include migration and invasion of glioma cells, biomarkers and radiosensitization.
    Keywords: Brain Neoplasms -- Metabolism ; Glioma -- Metabolism
    ISSN: 20450907
    E-ISSN: 2045-0915
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