Umfang:
1 Online-Ressource (23 Seiten)
ISSN:
1553-7374
Inhalt:
DCs express intrinsic cellular defense mechanisms to specifically inhibit HIV-1 replication. Thus, DCs are productively infected only at very low levels with HIV-1, and this non-permissiveness of DCs is suggested to go along with viral evasion. We now illustrate that complement-opsonized HIV-1 (HIV-C) efficiently bypasses SAMHD1 restriction and productively infects DCs including BDCA-1 DCs. ...
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In:
Public Library of Science, PLoS pathogens, Lawrence, Kan. : PLoS, 2005-, Band 11, Heft 6 (2015), 1553-7374
In:
volume:11
In:
year:2015
In:
number:6
Sprache:
Englisch
DOI:
10.1371/journal.ppat.1005005
URN:
urn:nbn:de:hebis:30:3-380087
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