1659486513

Bacterial Activation of Type I Interferons / edited by Dane Parker

Englisch

Cham : Springer, 2014

Online-Ressource (VII, 129 p. 12 illus., 10 illus. in color, online resource)

Druckausg.: Parker, Dane: Bacterial activation of type I interferons. - Cham : Springer, 2014. - VII, 129 S.

978-3-319-09498-4

Weitere Ausgaben: 978-3-319-09497-7 (Druckausgabe)

DOI: 10.1007/978-3-319-09498-4

*Bakterien / Aktivierung / Interferon

*Life sciences ; Immunology ; Emerging infectious diseases ; Bacteriology ; Life Sciences

Klassifikation der Library of Congress: QR74.8-99.5

Dewey Dezimal-Klassifikation: 579.3;

Regensburger Verbund-Klassifikation: WF 5000: W Biologie / WF 1000-WF 9600 Mikrobiologie / WF 5000-WF 8900 Bakteriologie / Allgemeines

Book Industry Communication: PSGD

bisacsh: SCI006000

The type I interferon (IFN) signaling pathway is well recognized as a pathway activated by viral infections. It is activated by a variety of microbial pattern recognition receptors including the Toll-like receptors, NOD-like receptors and several cytosolic receptors. Activation of the type I IFN pathway leads to the production of both antiviral factors and products that influence immune cell function. More recently it has been shown that bacteria are also capable of activating this pathway. Bacterial Activation of Type I Interferons reviews both the current understanding of how different bacterial species are able to activate this pathway as well as the influence type I IFNs have on the outcome to infection. Several different bacterial species are covered, spanning Gram positive and Gram negative, intracellular, extracellular, and different host infection sites. An introduction to the pathogenesis of each organism is provided, and the signaling molecules involved in the activation of the type I IFN pathway and the role it plays in animal infection models are also covered

Vervielfältigungen (z.B. Kopien, Downloads) sind nur von einzelnen Kapiteln oder Seiten und nur zum eigenen wissenschaftlichen Gebrauch erlaubt. Keine Weitergabe an Dritte. Kein systematisches Downloaden durch Robots.

https://doi.org/10.1007/978-3-319-09498-4 [E-books (Springer)]