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Umfang: 1 online resource (390 pages)

ISBN: 9780443154447 , 0443154449

Anmerkung: Front Cover -- Macrophomina Phaseolina -- Macrophomina Phaseolina -- Copyright -- Contents -- Contributors -- About the authors -- Preface -- I - The pathogen and disease -- 1 - Taxonomy of Macrophomina-traditional to molecular approaches -- 1.1 Introduction -- 1.2 History of Macrophomina phaseolina (Tassi) Goid -- 1.3 Molecular taxonomy of Macrophomina species -- 1.4 Host range and distribution of Macrophomina species -- 1.5 Intraspecific genetic diversity and pathogenic variability -- 1.6 Conclusions -- References -- 2 - Morphological and physiological characterization of Macrophomina isolates from Senegal -- 2.1 Introduction -- 2.2 Bio-ecology -- 2.3 Damage and economic importance of Macrophomina -- 2.4 Disease cycle -- 2.5 Characteristic of the isolates -- 2.5.1 Effect of temperature on the growth rate of Macrophomina sp. isolates -- 2.5.1.1 Macrophomina phaseolina -- 2.5.1.2 Macrophomina pseudophaseolina -- 2.5.1.3 Comparison of the two species at 36 and 40°C -- 2.5.2 Phenotype of the colonies -- 2.5.3 Sclerotia shape -- 2.5.4 Weight of sclerotia of both species -- 2.6 Conclusion -- References -- 3 - Macrophomina phaseolina causing various diseases in different crops -- 3.1 Introduction -- 3.2 Morphological characterization -- 3.3 Pathogenicity of M. phaseolina -- 3.4 Diseases caused by M. phaseolina -- 3.4.1 Charcoal rot -- 3.4.1.1 Hosts -- 3.4.1.2 Symptoms -- 3.4.1.3 Disease cycle -- 3.4.2 Damping-off -- 3.4.2.1 Hosts -- 3.4.2.2 Symptoms -- 3.4.2.2.1 Pre-emergence symptoms -- 3.4.2.2.2 Postemergence symptoms -- 3.4.2.3 Disease cycle -- 3.4.3 Stem blight -- 3.4.3.1 Hosts -- 3.4.3.2 Symptoms -- 3.4.3.3 Disease cycle -- 3.4.4 Leaf blight -- 3.4.4.1 Hosts -- 3.4.4.2 Symptoms -- 3.4.4.3 Disease cycle -- 3.4.5 Pod rot -- 3.4.5.1 Hosts -- 3.4.5.2 Symptoms -- 3.4.5.3 Disease cycle -- 3.5 Conclusion -- References. , 4 - Charcoal rot: a major root and stalk disease of sorghum -- 4.1 Introduction -- 4.2 Symptoms and signs -- 4.3 Pathogen biology -- 4.4 Infection process, disease cycle, and epidemiology -- 4.5 Disease control -- 4.5.1 Host resistance -- 4.5.2 Defense responses and molecular mechanisms of resistance -- 4.5.3 Cultural controls and nutrition -- 4.5.4 Fungicides -- 4.5.5 Biocontrol -- 4.6 Conclusions -- Acknowledgments -- References -- 5 - Recent advancement in management of soybean charcoal rot caused by Macrophomina phaseolina -- 5.1 Introduction -- 5.2 General information on soybean charcoal rot disease -- 5.2.1 Symptomatology -- 5.2.2 Disease cycle and epidemiology -- 5.2.3 Economic impact -- 5.3 Screening for charcoal rot resistance -- 5.3.1 Field screening -- 5.3.2 Artificial screening -- 5.4 Management of soybean charcoal rot -- 5.4.1 Biological control -- 5.4.2 Chemical control -- 5.4.3 Botanicals or plant extracts -- 5.4.4 Elicitors -- 5.4.5 Cultural management -- 5.4.6 Genetic resistance -- 5.5 Diagnosis of soybean charcoal rot and utilization in resistance identification -- 5.6 Host-pathogen interaction -- 5.7 Conclusions -- References -- 6 - Management of nematode-charcoal rot disease complexes in legumes -- 6.1 Introduction -- 6.2 Disease complexes of legumes -- 6.3 Management of disease complexes -- 6.3.1 Chemical control -- 6.3.2 Physical methods -- 6.3.3 Breeding for resistance -- 6.3.4 Biological control -- 6.3.5 Culture practices -- 6.3.6 Integrated management -- 6.4 Conclusions -- References -- 7 - Epidemiology and management of charcoal rot of maize caused by Macrophomina phaseolina (Tassi.) Goid -- 7.1 Introduction -- 7.2 Symptoms -- 7.3 Economic importance -- 7.4 The pathogen -- 7.5 Host range -- 7.6 Epidemiology -- 7.7 Disease cycle -- 7.8 Phytopathometry -- 7.9 Disease management -- 7.10 Conclusion -- References. , 8 - Spatial distribution of Macrophomina phaseolina (Tassi) Goid. in the Carpathian Basin and its damage on sunflowers -- 8.1 Introduction -- 8.1.1 Spread of M. phaseolina in europe -- 8.1.2 Yield losses caused by Macrophomina phaseolina -- 8.1.3 European countries -- 8.1.4 Oil content -- 8.1.5 Environmental conditions for disease development -- 8.1.6 Temperature -- 8.1.7 Soil moisture -- 8.1.8 Host plant and previous cropping -- 8.1.9 Crop rotation -- 8.1.10 Tillage -- 8.1.11 Irrigation -- 8.1.12 Chemical control -- 8.1.13 Biocontrol agents -- 8.1.14 Climate change -- 8.1.15 Hungarian climate -- 8.2 Materials and methods -- 8.2.1 Collection of isolates -- 8.2.2 Size of microsclerotia -- 8.2.3 Symptomatology -- 8.2.4 The survey -- 8.2.5 Description of sunflower hybrid -- 8.2.6 Weather data -- 8.2.7 Data analysis -- 8.3 Results -- 8.3.1 The survey of M. phaseolina in Hungary and in the Carpathian Basin -- 8.3.2 The size of microsclerotia -- 8.3.3 Symptoms on sunflower -- 8.3.4 Yield loss caused in Hungary -- 8.3.5 Effect of weather conditions on infection -- 8.3.6 Effect yield loss -- 8.3.7 Economic damage -- 8.4 Conclusion -- Acknowledgment -- References -- II - Resistance and pathogenicity -- 9 - Candidate genes of Macrophomina phaseolina involv -- 9.1 Introduction -- 9.2 Morphology characterization and life cycle of M. Phaseolina -- 9.2.1 Morphological characteristics -- 9.2.2 Genetic diversity -- 9.2.3 Life cycle of M. phaseolina -- 9.3 Crops and M. phaseolina interaction for disease development -- 9.4 M. Phaseolina enzymes and their role in pathogenicity -- 9.4.1 Enzymes produced by M. phaseolina -- 9.5 Molecular techniques to identify the pathogen specific genes -- 9.5.1 Molecular approach for identification of pathogenicity related genes -- 9.5.1.1 Markers and probes -- 9.5.1.2 Genomic approach -- 9.5.1.3 Quantitative PCR. , 9.5.1.4 Proteomic approach -- 9.5.1.5 CRISPER/CAS -- 9.5.1.6 Secretome analysis -- 9.6 M. phaseolina genes and their role in pathogenicity -- 9.7 Conclusion -- References -- 10 - Mechanism of disease development in host plants by Macrophomina phaseolina -- 10.1 Introduction -- 10.2 Survival of M. phaseolina -- 10.3 Genetic diversity of M. phaseolina -- 10.4 Microbial interactions in rhizosphere -- 10.4.1 Interaction with bacteria -- 10.4.2 Interaction with fungi -- 10.4.3 Interaction with mycorrhizal fungi -- 10.4.4 Interaction with nematodes -- 10.5 Pathogenesis by M. phaseolina -- 10.5.1 Role of inoculum in pathogenesis -- 10.5.1.1 Sclerotia density and germination -- 10.5.1.2 Factors affecting sclerotia germinations -- 10.5.1.3 Temperature -- 10.5.1.4 Soil moisture -- 10.5.1.5 Organic amendments -- 10.5.1.6 Inorganic amendments -- 10.5.1.7 Chemicals fungicides -- 10.5.2 Identification of host and adhesion -- 10.5.3 Induction of defense mechanism in host -- 10.5.4 Countering of host defense responses -- 10.5.5 Invasion -- 10.5.6 Establishment of disease -- 10.5.7 Symptoms caused by M. phaseolina -- 10.6 Role of cell wall-degrading enzymes (CWDEs) of M. phaseolina in pathogenesis -- 10.6.1 Pectin-degrading enzymes -- 10.6.2 Cellulose-hydrolyzing enzymes -- 10.6.3 Lignin-degrading enzymes -- 10.6.4 Protein-degrading enzymes -- 10.6.5 Hemicellulose-degrading enzymes -- 10.7 Role of toxins in pathogenesis by M. phaseolina -- 10.8 Macrophomina-host plants interaction -- 10.9 Conclusion -- References -- 11 - Molecular tools for detection and management of Macrophomina phaseolina -- 11.1 Introduction -- 11.2 Symptomatology -- 11.3 Causal organism -- 11.4 Disease cycle -- 11.5 Molecular identification of the pathogen -- 11.6 Internal transcribed spacer -- 11.7 PCR-finger printing techniques -- 11.8 Genetic diversity -- 11.9 SSR markers -- 11.10 RFLP. , 11.11 Genome sequencing -- 11.12 Metagenomics -- 11.13 Fluorescence in situ hybridization -- 11.14 Target sequences used for diagnostic assay development -- 11.15 Conclusion and future prospects -- References -- 12 - Genetics of plant resistance against Macrophomina phaseolina -- 12.1 Introduction -- 12.2 Pathogen background -- 12.3 Disease cycle -- 12.4 Symptomatology -- 12.5 Pathogenic and genetic variability -- 12.6 Genetic resistance -- 12.7 Management strategies -- 12.7.1 Cultural practices -- 12.7.2 Fungicides -- 12.7.3 Biological control -- 12.7.4 Botanicals -- 12.8 Conclusion -- References -- III - Ecology and epidemiology -- 13 - Ecological niche modeling of Macrophomina phaseolina (Tassi) Goid. using bio-climatic and non-bio-climatic var ... -- 13.1 Introduction -- 13.2 Material and methods -- 13.2.1 Data collections -- 13.2.2 Bio-climatic and non-bio-climatic variables -- 13.2.3 Habitat heterogeneity index -- 13.2.4 Land cover -- 13.2.5 Soil qualities -- 13.3 Data processing -- 13.3.1 Issue of multicollinearity -- 13.3.2 Species distribution modeling -- 13.3.3 Changes in core distribution centers and area loss and gain -- 13.3.4 Raster similarity analysis -- 13.4 Post maxent analysis -- 13.4.1 Niche overlap, range, and breadth -- 13.4.2 Random forest clustering (RF-C) -- 13.4.3 Uni-dimensional reliability analysis -- 13.5 Result and discussion -- 13.5.1 Dataset and issue of multi-collinearity -- 13.5.2 Maxent analysis -- 13.5.2.1 Current bio-climatic BC scenario -- 13.5.2.2 Bio-climatic-2050 scenario -- 13.5.2.3 Bio-climatic-2070 scenario -- 13.5.2.4 Habitat heterogeneity index -- 13.5.2.5 Soil factors -- 13.5.2.6 Land cover -- 13.5.3 Changes in core distribution centers and area loss and gain -- 13.5.4 Raster similarity analysis -- 13.5.5 Niche overlap, range, and breadth -- 13.5.6 Random forest clustering. , 13.5.7 Unidimensional reliability analysis.

Weitere Ausg.: Print version: Kumar, Pankaj Macrophomina Phaseolina San Diego : Elsevier Science & Technology,c2023 ISBN 9780443154430

Sprache: Englisch