UID:
edocfu_9958076426802883
Format:
1 online resource (144 p.)
Edition:
1st ed.
ISBN:
1-280-21036-2
,
9786610210367
,
0-309-56933-8
Series Statement:
National Academy of Sciences colloquium series
Note:
Bibliographic Level Mode of Issuance: Monograph
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COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935-1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS -- SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELS -- SODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS? -- CONCLUSIONS -- Calcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY -- FUNCTIONAL MRI STUDIES IN MONKEYS -- The spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY.
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ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS -- Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptors -- CONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord -- CONCLUSIONS -- The postnatal development of spinal sensory processing -- Transcriptional and posttranslational plasticity and the generation of inflammatory pain -- STATE-DEPENDENT SENSORY PROCESSING IN THE SOMATOSENSORY SYSTEM -- CONCLUSION -- Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions -- Does a neuroimmune interaction contribute to the genesis of painful peripheral neuropathies? -- Distinct neurochemical features of acute and persistent pain -- PREPROTACHYKININ-A PRODUCTS AND ACUTE PAIN -- PROTEIN KINASE C Γ AND PERSISTENT PAIN -- The genetic mediation of individual differences in sensitivity to pain and its inhibition -- THE SCOPE OF INDIVIDUAL VARIABILITY TO PAIN AND ANALGESIA -- HERITABILITY OF PAIN-RELATED TRAITS -- GENETIC CORRELATIONS AMONG PAIN PHENOTYPES -- IDENTIFICATION OF PAIN-RELATED GENES -- SEX-SPECIFIC GENETIC MEDIATION OF PAIN AND ANALGESIA -- ALLELIC VARIANTS OF PAIN-RELATED GENES.
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FUTURE DIRECTIONS -- The u opiate receptor as a candidate gene for pain: Polymorphisms, variations in expression, nociception, and opiate… -- NAS COLLOQUIA -- SCHEDULED -- COMPLETED -- 1999 ORDER FORM.
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English
Additional Edition:
ISBN 0-309-06548-8
Language:
English
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